23, 22 Calling the Microbiota to Control Atherosclerosis

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23, 22 Calling the Microbiota to Control Atherosclerosis. / Carreras, Alba; Bäckhed, Fredrik.

In: Immunity, Vol. 49, No. 5, 2018, p. 788-790.

Research output: Contribution to journal › Journal article › Research › peer-review

Harvard

Carreras, A & Bäckhed, F 2018, '23, 22 Calling the Microbiota to Control Atherosclerosis', Immunity, vol. 49, no. 5, pp. 788-790. https://doi.org/10.1016/j.immuni.2018.11.006

APA

Carreras, A., & Bäckhed, F. (2018). 23, 22 Calling the Microbiota to Control Atherosclerosis. Immunity, 49(5), 788-790. https://doi.org/10.1016/j.immuni.2018.11.006

Vancouver

Carreras A, Bäckhed F. 23, 22 Calling the Microbiota to Control Atherosclerosis. Immunity. 2018;49(5):788-790. https://doi.org/10.1016/j.immuni.2018.11.006

Author

Carreras, Alba ; Bäckhed, Fredrik. / 23, 22 Calling the Microbiota to Control Atherosclerosis. In: Immunity. 2018 ; Vol. 49, No. 5. pp. 788-790.

Bibtex

@article{a6312063978d4a6dbd1c40a2491f03a2,
title = "23, 22 Calling the Microbiota to Control Atherosclerosis",
abstract = "While the commensal microbiota is considered an important regulator of immunity and metabolism, the mechanisms controlling the interplay between diet, cytokine signaling, and the microbiota in atherosclerosis remains unknown. In this issue of Immunity, Fatkhullina et al. (2018) demonstrate that interlukin-23-22 axis regulates diet-induced atherosclerosis by repressing pro-atherogenic microbiota.",
author = "Alba Carreras and Fredrik B{\"a}ckhed",
year = "2018",
doi = "10.1016/j.immuni.2018.11.006",
language = "English",
volume = "49",
pages = "788--790",
journal = "Immunity",
issn = "1074-7613",
publisher = "Cell Press",
number = "5",

}

RIS

TY - JOUR

T1 - 23, 22 Calling the Microbiota to Control Atherosclerosis

AU - Carreras, Alba

AU - Bäckhed, Fredrik

PY - 2018

Y1 - 2018

N2 - While the commensal microbiota is considered an important regulator of immunity and metabolism, the mechanisms controlling the interplay between diet, cytokine signaling, and the microbiota in atherosclerosis remains unknown. In this issue of Immunity, Fatkhullina et al. (2018) demonstrate that interlukin-23-22 axis regulates diet-induced atherosclerosis by repressing pro-atherogenic microbiota.

AB - While the commensal microbiota is considered an important regulator of immunity and metabolism, the mechanisms controlling the interplay between diet, cytokine signaling, and the microbiota in atherosclerosis remains unknown. In this issue of Immunity, Fatkhullina et al. (2018) demonstrate that interlukin-23-22 axis regulates diet-induced atherosclerosis by repressing pro-atherogenic microbiota.

U2 - 10.1016/j.immuni.2018.11.006

DO - 10.1016/j.immuni.2018.11.006

M3 - Journal article

C2 - 30462992

VL - 49

SP - 788

EP - 790

JO - Immunity

JF - Immunity

SN - 1074-7613

IS - 5

ER -

ID: 209354525